Transmural infarction: haddasawa da kuma hangen nesa

Transmural infarction - mai kowa da matukar hatsari cutar, wanda yana tare da necrosis daga cikin zuciya tsoka. Ya kamata a lura da cewa mace-mace kudi a taron na kama Pathology ne sosai high, musamman idan ãyõyin cardiac cuta da aka ba su gani ba a lokacin, da kuma haƙuri kuwa ba ta karɓe da zama dole kiwon lafiya. Saboda haka da muhimmanci a san yadda za a bayyana wannan nau'i na ciwon zuciya da kuma abin da ake bukata taimako ga marasa lafiya da mutum.

Transmural tsokar zuciya infarction: abin da yake da shi?

Da farko, bari mu magance da ma'anar kalma. Lalle ne mãsu yawa daga gare ku sani cewa infarct yanayi suna tare da withering bãya daga rabo daga cikin zuciya tsoka. Wannan ya faru saboda zato lõkacin fatara daga jini ya kwarara zuwa cikin zuciya, sakamakon nama ba sama da zama dole ga rai na oxygen da kuma gina jiki.

Kamar yadda aka sani, zuciya bango kunshi uku yadudduka - a epicardium (m Layer), mural murdede Layer da endocardium (na ciki na tsokoki). Lokacin da intramural form necrosis cutar rinjayar kawai guda Layer. M transmural tsokar - Pathology, inda necrotic matakai za a iya lura a duk yadudduka na zuciya. An ce, wannan shi ne mafi hatsari nau'i na wannan cuta.

A zamani magani, Pathology ne classified bisa sarrafawa na foci na necrosis. A mafi akai-akai lura transmural tsokar zuciya infarction gaban bango, wato hagu ventricle. Daya kawai daga biyar lokuta, necrosis da kuma rinjayar da dama ventricle bango. Game da na uku daga cikin marasa lafiya da hannu a cikin tsari da kuma ganuwar da atria.

A Sanadin cutar

A kusan kowace harka, da sakamakon transmural tsokar ischemic cututtukan zuciya. Kamar yadda aka ambata riga, tare da kasa oxygen da kuma gina jiki ga tsoka Kwayoyin fara mutuwa - don haka tasowa necrosis. Bisa kididdigar da, 90% na lokuta cikin hanyar shi ne atherosclerosis na jijiyoyin zuciya tasoshin, wanda damar jini ya kwarara zuwa cikin myocardium.

A kan bango na jirgin ruwa da aka kafa da ake kira atherosclerotic plaque. Ƙarƙashin rinjayar wasu dalilai plaques rabo tsattsãgẽwa daga bangon da ya rufe da lumen na jijiyoyin zuciya jijiya. M transmural tsokar yawanci progresses sauri a cikin wadanda yanayi a lokacin da tsokar oxygen bukatar ƙaruwa sharply - wannan na iya faruwa, misali, tare da karfi na zahiri aikinsa, zazzabi, m wani tunanin danniya.

M transmural tsokar zuciya infarction: Hadarin dalilai

Kamar yadda ka gani, necrotic raunuka na zuciya ba wani m cutar. A mafi yawan lokuta, da suka ci gaba a kan bango na riga data kasance atherosclerosis da ischemia. Saboda haka yana yiwuwa a ware da kuma da dama hadarin dalilai:

• A kayyade predisposition na haƙuri da ake dangantawa da wasu sifofin da kuma gaji cututtuka.
• Yana daukan wuri da shekaru factor. Transmural tsokar sau da yawa bincikar lafiya a mutane a kan 45, da kuma maza ne mafi yiwuwa ga wannan cuta.
• Karuwan matakin na cholesterol a cikin jini (saboda wannan nau'i plaques a kan garun jini, wanda baya rufe jini).
• M rage cin abinci (cin m da soyayyen abinci qara da alama na tasowa atherosclerosis).
• Kiba (wanda a mafi yawan lokuta ake dangantawa da matalauta da abinci da kuma nauyi riba qara load a kan zuciya).
• Jiki rashin aiki (sedentary salon kaiwa zuwa wani sauka a hankali weakening na zuciya tsoka, da myocardium bayan wanda yana da matukar wuya a magance da wani jiki aiki).
• Constant danniya, burnout, wani tunanin danniya take kaiwa zuwa hormonal canje-canje da zai shafi zuciya da jijiyoyin jini tsarin.
• Smoking (bisa ga statistics, 35% na lokuta da tsokar necrosis hade da wannan mummunan al'ada).

Main cututtuka: yadda za a gane mai ciwon zuciya?

A bayyanar cututtuka na necrosis iya zama daban-daban - shi duka ya dogara da mataki na lalacewa, da mutum ta shekaru, kasancewar concomitant cututtuka, da dai sauransu. Akwai lokuta da asymptomatic tsokar kwarara ... Duk da haka, wasu daga cikin manyan cututtuka su ne:

• A mafi yawan lokuta, da mutum tanã kai ƙãra na tsanani compressive zafi a baya da breastbone - shi a fili ba to hagu kafada, hannu, muƙamuƙi, kunne kuma ko da hakora.
• Pain wavy kuma dogon - hare-haren za a iya maimaita a cikin 'yan sa'o'i ko ma kwanaki.
• Tasowa tachycardia, da mãsu haƙuri yawanci ji sosai m, kuma ko da m balli.
• Akwai kuma ji na gajere Fading zuciya.
• Akwai m ãyõyin a zuciya hari - fata da kuma mucous membranes mutum abruptly kodadde.
• Kan bango na necrosis akai-akai ci gaba cardiac fuka - haƙuri lura da wani karfi shortness na numfashi, kwatsam hare-hare na breathlessness.

diagnostic matakan

Lokacin da na farko take hakkin ya kamata a hawa nan da nan zuwa ga rashin lafiya mutum a wani asibiti unguwa, inda dace ganewar asali da kuma magani ne da za'ayi. Babban Hatsari Hanyar ga tsokar necrosis ne electrocardiography, saboda lokacin da irin wannan hanya ne zai yiwu a gyara lantarki iko a sassa daban daban na zuciya.

Daidai da za'ayi, kuma decrypted electrocardiogram damar da likita domin sanin sarrafawa na necrosis (msl, ƙananan transmural ciwon zuciya), da mataki na yaduwar wani pathological aiwatar da rauni zurfin, duration da cutar.

Bugu da kari, kana bukatar wani jini gwajin. Tsokar lura karuwa erythrocyte sedimentation kudi, ta ƙara yawan leukocytes bayyana a cikin jini na musamman enzymes da mahadi wanda kullum ne a cikin tsokar cell.

Sharuddan taimakon farko

M transmural tsokar zuciya infarction gaban bango - a Pathology cewa tasowa sosai da sauri. Its bayyanar cututtuka zai iya faruwa a kusan kowane lokaci, don haka ya kamata ka familiarize kanka da dokoki na taimako ga mãsu haƙuri. Hakika, na farko na duk muna bukatar mu kira motar asibiti.

Jiran da likitoci, da mutum na bukatar kwanta. An togiya ne a halin da ake ciki inda a can ne mai karfi da nakasa daga 'ya'yan numfashi da kuma wheezing lokacin da numfashi jinsin - wanda idan da haƙuri shi ne mafi alhẽri wurin zama, tucked karkashin baya na matashin kai ko matashi. An shawarar ya dauki wani asfirin. Kana bukatar kuma ka ba da mutum nitroglycerin - na farko daya kwaya, kuma idan zafi ba ya tafi, sai bayan minti biyar wani daya (amma ba fiye da uku).

Students jiyya na tsokar

Dangane da samu sakamakon a lokacin da ganewar asali likita ta zayyana magani. Harufan magani yawanci kunshi uku main sassa:

• Yarda na maganin ciwo don ya rage haƙuri da yanayin. Popular sau da yawa, zafi ne mai yiwuwa a daina kawai da taimakon m kwayoyi, ciki har da "Promedol" da "Morphine". Bayyanar da bayyanar cututtuka da ganewar asali "tsokar zuciya infarction" ne mai danniya ga mutum, da tashin hankali da tsoro kawai qara nauyi a kan zuciya - da haƙuri da aka wajabta sedatives.
• A regimen kamata hada da samun nitrates da beta-blockers, wanda taimaka wajen kawar da arrhythmia da kuma mayar da zuciya kari.
• Lokacin da transmural tsokar high yiwuwa na jini clots, wanda shi ne fraught tare da necrosis na sauran gabobin da kuma yaduwar tsari a kan m yankunan na myocardium, juyayi da tsarin raunuka da kuma mutuwar ma. Saboda haka, da yaki da thrombosis kamata a fara a farkon sa'o'i bayan da farko na bayyanar cututtuka. A saboda wannan dalili, yi amfani da vasospasm shirye-shirye wanda dauke da heparin da fibrinolysin. Babu shakka, a lokacin da shan irin kwayoyi ne wata ila don m hemorrhage saboda jini gudan jini tsaya a nan ba, don haka yana da muhimmanci a hankali saka idanu da haƙuri da yanayin.

Magani ne dole ciyar a asibiti - haƙuri a kusa da Agogon ya zama karkashin wani likita ta kula.

A sakamakon da rikitarwa da cutar

Transmural infarction - mafi wuya da kuma m nau'i na tsokar necrosis, musamman a lokacin da ta je manyan mai da hankali necrosis. The rashin lafiya ne fraught da sakamakon - ci gaban da na huhu edema, inna da wata gabar jiki, jawabin cuta. Wani lokaci na tsokar entails kara Pathology na jijiyoyin bugun gini tsarin, kamar bugun jini.

A sakamakon wannan cuta kuma sun hada da thromboembolism, na ramin zuciya fibrillation, ƙarshe na aikin daban-daban hukumomin har da tsarin. Irin haka sau da yawa ƙare a cikin mutuwa, ko idan haƙuri likita taimako da aka bayar. Wani rai-barazanar rikitarwa - ciwon zuciya. Idan wani karfi rage daga cikin myocardium da kuma kara matsa lamba a cikin ventricles da atria hadarin compromising da mutunci da tsokoki a yankin na necrosis.

Gyaran fuska da kuma kintace

Abin baƙin ciki, kintace a kan irin likita yanayi ba ma m - game da 50% daga cikin lokuta (musamman a lokacin da ya zo ga tsofaffi) kashe. Idan, duk da haka, har yanzu gudanar don maido da jini ya kwarara, akwai har yanzu a hadarin rikitarwa a nan gaba, tun can sun kasance canje-canje na tsokar nama.

Wannan shi ne dalilin da ya sa yana da muhimmanci ne fi lokaci. Marasa lafiya suna shawarar da sauki, sauki, da kuma high-kalori abinci. Har ila yau, yana da muhimmanci a hankali sake samu locomotor aiki, kamar yadda rashin aiki ne fraught tare da atrophy na tsokoki da kuma m, a cikin huhu. Hakika, dukkan ayyukan da suka shafi jiki aiki, ya kamata a tsananin sarrafawa gwani.

Taimaka far da gudana monitoring da wani likita ne zama dole kafin karshen rayuwa. Hakika, marasa lafiya bukatar bi rage cin abinci, guje wa danniya da kuma zafin jiki, kauce wa m karuwa jini, da kuma daina shan taba.