Kiwon lafiyaMagani

Atherosclerosis

Atherosclerosis, a tsakanin sauran cututtuka na gefe jini yana da wani abu matsayi.

Mafi sau da yawa, da rashin lafiya rinjayar maza girmi shekara arba'in. Atherosclerosis ne sau da yawa a cikin hanyar m ischemia, ka hukunta marasa lafiya zuwa sosai azãba mai raɗaɗi. Duk da haka, da yawa marasa lafiya rasa su ikon yin aiki. Mafi yawa alama atherosclerosis na ƙananan reshe jijiyoyi. A tsari iya sarrafa a cikin lakã. Bugu da kari, sau da yawa cutar rinjayar srednekalibernye jijiya - femoral da popliteal.

Obliterating atherosclerosis na ƙananan extremities dangantaka da wannan etiological dalilai da pathogenetic sunadaran cewa fararwa da cutar wasu sarrafawa.

A manyan canje-canje suna lura a lipoidoza foci karkara. Wannan kiwata sabo connective nama. Maturing, shi take kaiwa zuwa girma na fibrous plaques. Wadannan tsarin subsidence auku fibrin da platelet clots. M sia jari tsokani wani cuta jini wurare dabam dabam a cikin plaques. Su necrosis sa, bi da bi, da samuwar atheroma. Dauke da cavities atheromatous taro fall a cikin lumen da jirgin ruwa. Tsage a distal shugabanci, za su iya tsokana embolism.

A lokaci guda, da fallasa su canje-canje a cikin tsokoki na plaques a cikin yankunan na roba zaruruwa sha degeneration, fara sa alli salts. Kamar wancan atherosclerosis fitowa a karshe mataki.

A lokacin da dukan da cutar ne hudu, saukarwa.

Atherosclerosis da shekaru da yawa na iya ci gaba ba tare da wani bayyanar cututtuka. Duk da haka, tun farko na asibiti cututtuka sau da yawa progresses sauri sosai.

A wasu lokuta, a cikin ra'ayi na acceding thrombosis, cuta bayyananen halin suddenness. A tarihin sau da yawa a cikin marasa lafiya tare da hauhawar jini, tarihi na tsokar, angina, ciwon sukari, Sistem sakulasan cuta na kwakwalwa.

A bayyanar cututtuka da cutar kamata a classified a matsayin intermittent claudication, wanda aka bayyana soreness na maraƙi tsokoki. A majiyai yawanci faruwa a lokacin tafiya, kuma bace bayan wani gajeren hutawa.

Atherosclerotic raunuka na iliac arteries da m rabo daga ciki lakã tsokane sarrafa zafi ba kawai a cikin shins, amma gluteal cinya tsokoki kazalika a cikin lumbar yankin. Karfafa na intermittent claudication lura a lokacin Uphill ko matakala.

Hankula bayyanar cututtuka suna dauke su a gabar nutsuwa, kiwon kafafu ji na ƙwarai to sanyi, kangararre ƙafafunsa. A sakamakon ischemia akwai wani canji na launi daga cikin ƙananan extremities. A farkon ci gaban da cuta, su zama kodadde a launi. A cikin shakka daga cutar fata na yatsunsu da ƙafãfunsu zama m-cyanotic.

Trophic cuta rushe ci gaban da kusoshi taimakawa wajen gashi hasara.

Ci gaban da cutar halin ulcerative-necrotic canje-canje a cikin taushi tsokoki na distal zones da shafi gabar jiki. An lura da edema da hyperemia tasha. Aortoiliac occlusion rabo ne mai halayyar alama na rashin ƙarfi sa da cuta a cikin jini wurare dabam dabam.

Jarrabawa da marasa lafiya sau da yawa ya nuna atrophy ko wasting na tsokoki a cikin ƙananan extremities.

Sau da yawa akwai shan kashi na femoropopliteal kashi, wanda aka nuna a cikin rashi na wani ripple a kan arteries na tasha ko popliteal jijiya daga batu na asalin da zurfin femoral jijiya.

Babban Hanyar ganewar asali ne dauke angiography. Wannan hanya damar su gane da irin nisan da kuma sarrafawa da pathological tsari, da matakin da jijiyoyin bugun gini raunuka, halin na biyu dabam dabam, kazalika don kimanta Jihar jini ya kwarara distal tashar.

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