Bai cika dama cuta reshe block

A wannan labarin, za mu duba wannan Pathology kamar yadda bai cika kawancen na da hakkin kafa cuta reshe block. Wannan juzu'ine na take hakkin lantarki hasken dake fitowa a cikin zuciya. Wannan cuta ne yawanci asymptomatic, amma shaida kwayoyin cututtukan zuciya da kuma yin tasiri cikin sakamako na farko da cutar. A ganewar asali dogara ne a kan sakamakon electrocardiographic karatu. Bai cika dama cuta reshe block ne a daya da rabi zuwa biyu duka hudu-tenths bisa dari na lokuta, kuma tare da shekaru, wadannan Figures kawai kara. Daga cikin iri daban-daban blockades a mita wannan Pathology aka ranked biyu.

Etiology na da ya faru

A dalilin iya zama da zuciya lahani, jijiyoyin zuciya jijiya cuta da wani yawan abin sama na anti-arrhythmia, na huhu zuciya, stenosis na mitral bawul, na huhu hauhawar jini, tricuspid zuciya bawuloli, m tsokar zuciya infarction, myocarditis, cardio, da hawan jinni zuciya, rashin daidaituwa daga Wutan a cikin jiki, da kuma dama na ramin zuciya hypertrophy. Quite rare bai cika dama cuta reshe block kamu a lafiya matasa. A irin haka ne, muna magana ne game da ne na al'ada.

Bai cika dama cuta reshe block ne cikin hanyar deceleration aika da biyu sinus da ectopic hatsaisai a kanta. Wannan, bi da bi, take kaiwa zuwa wani m canji a cikin zumudi jerin wasu daga cikin sassa na ventricles ko kawai gaba daya ventricle.

Bai cika kawancen dama cuta reshe block: bayyanar cututtuka

A mafi yawan lokuta, wannan Pathology ba ya haifar da wani m bayyanar cututtuka, saboda da kari kuma mita na zuciya ba ya canzawa.

Ganewar asali da wannan cuta, kamar yadda aka ambata a sama, dogara ne a kan data samu ta hanyar electrocardiographic karatu.

Ga jerin alamun da ke bayyane a kan shi:

• QRS hadaddun duration ne ta ƙara dan kadan, har zuwa tara ko goma sha hundredths na biyu ne.
• A cikin farko da V6 kaiwa dan kadan widened hakori S da dama da nono ayata1 sata QRS hadaddun wakiltar rSr, ko rsR.

Bai cika kawancen dama cuta reshe block: magani

A tsawon lokaci da kuma kwanciyar hankali na data kasance Pathology sau da yawa ba ya bukatar wani magani. Amma a yanayi inda ta bayyanar daidai da ƙarin tsanani na cututtukan zuciya, dace magani ne na bayar da muhimmanci ga daidaita cardiac madugu. Idan kafafuwansa biyu kawancen m, wucin gadi pacing nuna, bayar da kara duration na HV-tazara kan 80-100 ƴan daƙiƙa. All mazan marasa lafiya da madugu munanan irin wannan ne batun wajibi asibiti. A m cuta da kuma a cikin rashi na contraindications miyagun ƙwayoyi far za a iya amfani da, ba tare da ya manta inconstancy da unreliability na wannan wajen tasiri wannan Pathology. A cikin mafi kyau idan, za ka iya ƙidaya a kan wani wucin gadi sakamako.

Suna amfani domin lura da irin kayayyakin:

1. Anticholinergics, wanda rage vagal tsoma, msl 0.1% atropine daya milliliter intravenously da kuma 0.2% platifillin matsayin daya milliliter ko kamar yadda subcutaneously, intravenously, amma a wannan yanayin wajibi ne a gabatar da 5% glucose a juz'i na zuwa 500 ml.
2. Formulations reinforcing sakamako daga cikin m cardiac madugu tsarin, misali, 5% ephedrine daya milliliter intramuscularly, subcutaneously, ko intravenously, 0.2% norepinephrine, a cikin juz'i na 1 ml, an gudanar a kan glucose ko 0.05% alupent rabin daya shigen sukari, gudanar biyu intramuscularly da intravenously, ko 0.1% izadrin a daya milliliter girma.
3. Kamar yadda amfani glucocorticosteroid hormones, msl, hydrocortisone, a wani kudi na har zuwa 200 MG da rana.
4. Don rage abun ciki na potassium da kyawawa don amfani 1% na Lasix a daya milliliter yawa.

Hangen nesa da wannan cuta ne quite m, amma, ba hadarin muni su dukan related cardiac cututtuka, dole ne ƙwarai da gaske ga magani.