Na ramin zuciya wanda bai kai lashe: shi kuma ya faru a al'ada

A wata al'ada zuciya rate kafa musamman sinoatrial kumburi, wanda za a iya samu a dama atrium. Ya saki wani cajin da wutar lantarki da cewa yana sa da atria zuwa mu'ãmalar. Ya ta wani hadadden tsarin na musamman Kwayoyin rarraba tare da myocardium. Frequency na contractions da kayyade a al'ada jijiyoyi da takamaiman humoral (catecholamines, msl, epinephrine). Saboda haka da zuciya adapts da bukatun da rundunar kwayoyin, wato, a lokacin da sau na danniya, tashin hankali ko ta jiki exertion mita rate zama yawa ya fi girma.

Beats - wani "wedging" na ƙarin tunkaro ga al'ada kari na zuciya, su ne - wani matsanancin zuciya da haifar da ƙarin matsaloli. Da suka faru a lokacin da wani lantarki cajin da aka canjawa wuri daga yankin waje da sinoatrial kumburi.

Supraventricular arrhythmias faruwa a biyu lokuta. Ko idan wasu yanki na atrium aka depolarized gaba da lokaci, ko idan m lokacinta aika atrioventricular kumburi. A 60% na lafiya mutane akwai mutum lashe "haife" daga atrium. Duk da haka, suna da hankula ga wani yanayi, kamar ciwon zuciya, da kuma munanan na mitral bawul. Suna iya fararwa da uncontrolled ƙanƙancewa - fibrillation, sabili da haka ba za a iya watsi da esktrasistoly. Barasa tare da maganin kafeyin exacerbate matsalar.

Na ramin zuciya wanda bai kai lashe faru a mutane na kowane zamani. Shi ne ba a duk nadiri. Idan ECG rikodin for 24 hours, sa'an nan 63% na cikakken lafiya cardiolog gano guda PVCs. Duk da haka, babban adadin su da ake samu a mutane da mahaukaci zuciya tsarin. Wannan ya faru sau da yawa bayan wani ciwon zuciya.

Yara na ramin zuciya arrythmia faruwa game da yadda sau da yawa kamar yadda atrial yawanci isa yau da kullum motsa jiki na low tsanani cewa shi ya daina bayyana. Kawai a yanayin saukan mahaukaci tsarin da atria, shi zai iya sa atrial fibrillation.

Amma ga zuciya, wanda yake shi ne daidai tsari, shi dole ne a ce, ba hatsari na ramin zuciya arrythmia. Duk da haka, idan suka fara bayyana more sau da yawa a lokacin motsa jiki, yana da wani mummunan alama.

Idan gano abnormality na tsarin, ya kamata ziyarci wani likitan zuciyar. In ba haka ba halin da ake ciki zai iya zama barazana ga rayuwa.

Hadarin dalilai bayyananen lashe hada da hauhawar jini, balagagge shekaru, na ramin zuciya hypertrophy, ciwon zuciya, cardiomyopathy, rashi na alli, magnesium da potassium, amphetamine, tricyclic antidepressants, kuma digoxin, barasa zagi, danniya, maganin kafeyin ci, da kuma cututtuka.

Yawanci, marasa lafiya koka palpitations. Na ramin zuciya wanda bai kai lashe bayyana m ragi bayan al'ada tasiri ya kuma bi da nufi da "dakatar" da zuciya. Shi ne mai ban ji ga wani mutum wanda ba a sani, don haka shi ne lura a matsayin alama. Wasu mutane a lokaci guda sosai damu.

Kullum kadai yanayin worsens, kuma a load - kara da kanta. Duk da haka, idan kaya suna samun karfi, yana da ba mai kyau alama.

Har ila yau, bayyanar cututtuka suna rufe da mãgãgi, wani rauni, kuma kullum tari cewa ba za a iya bayyana ta sauran haddasawa.

Waɗanda suka yi zargin cardiac anomalies Tsarin sanya echocardiography da kuma duban dan tayi. Cak na jini da kuma adadin thyroid hormone, kazalika da isar a cikin jini electrolyte (alli, magnesium, potassium). Sau da yawa samfurori da ake gudanar a tilasta jiki aiki: mita extrasystoles bari a karkashin load da kuma a sauran.

A mafi yawan lokuta beats - ba wani dalilin ƙararrawa, amma idan ka zargin su a - je zuwa wani likita ka amince. Constant tashin hankali da aka kashe fiye da mutane fiye da lashe.